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Wanleibio rabbit anti cox2 antibody
Rabbit Anti Cox2 Antibody, supplied by Wanleibio, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+anti+cox2+antibody/anti+cox2+rabbit/pm42132245-175-0-6
Average 86 stars, based on 1 article reviews
rabbit anti cox2 antibody - by Bioz Stars, 2026-09
86/100 stars

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Related Articles

Virus:

Article Title: MNS induces antiviral protection and suppresses inflammation.
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Article Title: MNS induces antiviral protection and suppresses inflammation
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Enzyme-linked Immunosorbent Assay:

Article Title: MNS induces antiviral protection and suppresses inflammation.
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Article Title: MNS induces antiviral protection and suppresses inflammation
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Infection:

Article Title: MNS induces antiviral protection and suppresses inflammation.
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Article Title: MNS induces antiviral protection and suppresses inflammation
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Expressing:

Article Title: MNS induces antiviral protection and suppresses inflammation.
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Article Title: MNS induces antiviral protection and suppresses inflammation
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Quantitative RT-PCR:

Article Title: MNS induces antiviral protection and suppresses inflammation.
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.

Article Title: MNS induces antiviral protection and suppresses inflammation
Article Snippet: Rabbit anti-COX2 antibody was obtained from Wanleibio.



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Metabolites suppress the progression of OvE in vivo (A) Schematic representation of experimental timeline and procedures in rat OvE model. This figure was created in BioRender ( https://BioRender.com/rlasz0x ). (B) The normal ovaries and OvE lesions from NC, GA, AA, and HA groups. The masses of lesions from the indicated treatment groups are shown. Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05. (C) Expression of endometriosis-related cytokines in OvE tissues were analyzed by qPCR. The mRNA levels are expressed relative to transcript level in normal ovary tissue, set at 1.0 ( n = 5 per group). Asterisks indicate significance between groups: ∗ p < 0.05; (D) Representative H&E-stained sections of lesions and sections stained <t>with</t> <t>anti-COX2</t> (inflammation), anti-Ki67 (proliferation), anti-F4/80 (macrophages and inflammation), and anti-VEGFA (angiogenesis). Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001. Scale bars, 100 mm.
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Role of ferroptosis in APAP-induced liver injury in C57BL/6J mice (A) IHC staining of GPX4 (upper panel) and <t>PTGS2</t> (middle panel) proteins and TUNEL staining (bottom panel) in hepatocytes of C57BL/6J mice treated with APAP ± BAZ at indicated doses for 24 h. For each staining, the upper images represent the original low-magnification views, and the lower images show the corresponding enlarged areas. Scale bars = 1 mm (upper images) and 250 μm (lower images). (B–D) The respective quantitative intensity values of GPX4 protein level (B, n = 3), PTGS2 protein level (C, n = 3), and TUNEL staining level (D, n = 3). All quantitative data are presented as mean ± S.D. (∗∗ or ## p < 0.01).
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Role of ferroptosis in APAP-induced liver injury in C57BL/6J mice (A) IHC staining of GPX4 (upper panel) and <t>PTGS2</t> (middle panel) proteins and TUNEL staining (bottom panel) in hepatocytes of C57BL/6J mice treated with APAP ± BAZ at indicated doses for 24 h. For each staining, the upper images represent the original low-magnification views, and the lower images show the corresponding enlarged areas. Scale bars = 1 mm (upper images) and 250 μm (lower images). (B–D) The respective quantitative intensity values of GPX4 protein level (B, n = 3), PTGS2 protein level (C, n = 3), and TUNEL staining level (D, n = 3). All quantitative data are presented as mean ± S.D. (∗∗ or ## p < 0.01).
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Role of ferroptosis in APAP-induced liver injury in C57BL/6J mice (A) IHC staining of GPX4 (upper panel) and <t>PTGS2</t> (middle panel) proteins and TUNEL staining (bottom panel) in hepatocytes of C57BL/6J mice treated with APAP ± BAZ at indicated doses for 24 h. For each staining, the upper images represent the original low-magnification views, and the lower images show the corresponding enlarged areas. Scale bars = 1 mm (upper images) and 250 μm (lower images). (B–D) The respective quantitative intensity values of GPX4 protein level (B, n = 3), PTGS2 protein level (C, n = 3), and TUNEL staining level (D, n = 3). All quantitative data are presented as mean ± S.D. (∗∗ or ## p < 0.01).
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Image Search Results


Metabolites suppress the progression of OvE in vivo (A) Schematic representation of experimental timeline and procedures in rat OvE model. This figure was created in BioRender ( https://BioRender.com/rlasz0x ). (B) The normal ovaries and OvE lesions from NC, GA, AA, and HA groups. The masses of lesions from the indicated treatment groups are shown. Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05. (C) Expression of endometriosis-related cytokines in OvE tissues were analyzed by qPCR. The mRNA levels are expressed relative to transcript level in normal ovary tissue, set at 1.0 ( n = 5 per group). Asterisks indicate significance between groups: ∗ p < 0.05; (D) Representative H&E-stained sections of lesions and sections stained with anti-COX2 (inflammation), anti-Ki67 (proliferation), anti-F4/80 (macrophages and inflammation), and anti-VEGFA (angiogenesis). Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001. Scale bars, 100 mm.

Journal: iScience

Article Title: Serum metabolic fingerprinting for diagnosis and therapeutic applications of ovarian endometriosis

doi: 10.1016/j.isci.2026.114887

Figure Lengend Snippet: Metabolites suppress the progression of OvE in vivo (A) Schematic representation of experimental timeline and procedures in rat OvE model. This figure was created in BioRender ( https://BioRender.com/rlasz0x ). (B) The normal ovaries and OvE lesions from NC, GA, AA, and HA groups. The masses of lesions from the indicated treatment groups are shown. Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05. (C) Expression of endometriosis-related cytokines in OvE tissues were analyzed by qPCR. The mRNA levels are expressed relative to transcript level in normal ovary tissue, set at 1.0 ( n = 5 per group). Asterisks indicate significance between groups: ∗ p < 0.05; (D) Representative H&E-stained sections of lesions and sections stained with anti-COX2 (inflammation), anti-Ki67 (proliferation), anti-F4/80 (macrophages and inflammation), and anti-VEGFA (angiogenesis). Data are presented as mean ± SEM ( n = 5). Asterisks indicate significance between groups: ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001. Scale bars, 100 mm.

Article Snippet: Anti-COX2 , Cell Signaling Technology , Cat# 12282, RRID: AB_2571729.

Techniques: In Vivo, Expressing, Staining

Role of ferroptosis in APAP-induced liver injury in C57BL/6J mice (A) IHC staining of GPX4 (upper panel) and PTGS2 (middle panel) proteins and TUNEL staining (bottom panel) in hepatocytes of C57BL/6J mice treated with APAP ± BAZ at indicated doses for 24 h. For each staining, the upper images represent the original low-magnification views, and the lower images show the corresponding enlarged areas. Scale bars = 1 mm (upper images) and 250 μm (lower images). (B–D) The respective quantitative intensity values of GPX4 protein level (B, n = 3), PTGS2 protein level (C, n = 3), and TUNEL staining level (D, n = 3). All quantitative data are presented as mean ± S.D. (∗∗ or ## p < 0.01).

Journal: iScience

Article Title: Bazedoxifene rescues hepatocytes from chemically induced oxidative ferroptotic injury in vivo and in vitro by inhibiting protein disulfide isomerase

doi: 10.1016/j.isci.2026.114931

Figure Lengend Snippet: Role of ferroptosis in APAP-induced liver injury in C57BL/6J mice (A) IHC staining of GPX4 (upper panel) and PTGS2 (middle panel) proteins and TUNEL staining (bottom panel) in hepatocytes of C57BL/6J mice treated with APAP ± BAZ at indicated doses for 24 h. For each staining, the upper images represent the original low-magnification views, and the lower images show the corresponding enlarged areas. Scale bars = 1 mm (upper images) and 250 μm (lower images). (B–D) The respective quantitative intensity values of GPX4 protein level (B, n = 3), PTGS2 protein level (C, n = 3), and TUNEL staining level (D, n = 3). All quantitative data are presented as mean ± S.D. (∗∗ or ## p < 0.01).

Article Snippet: Cox2 (D5H5) XP® Rabbit mAb , Cell Signaling Technology , Cat#12282S; RRID: AB_2571729.

Techniques: Immunohistochemistry, TUNEL Assay, Staining